AIM To observe protective effect of hyperoxia balanced saline solution on reperfusion injury caused by bleeding shock. 目的观察高氧平衡盐液对防治家兔失血性休克再灌注损伤效果。
Conclusions: Transient exposure to hyperoxia at hyperbaric pressures prevented brain injury by inhibition of apoptosis. 结论：短暂高压氧治疗可以抑制细胞凋亡，防止脑损伤。
Combining the helium and hyperoxia slows down the frequency of breathing while making the air easier to breathe. 氦氧联用减慢了呼吸频率从而使空气更容易呼出。
Positive staining cells was seldom seen in the pulmonary tissues in rats in the hyperoxia dexamethasone group. 高氧组和地塞米松组肺组织除了较厚肺间隔偶见散在数个细胞显色外,整个肺组织几乎见不到阳性细胞。
After 7 days of exposure, stronger immunostaining for eNOS in the airway epithelium of the hyperoxia group than of the air group was seen. 与空气组比较 ;7d时高氧组气道上皮细胞eNOS表达增加 (P <0 .;0 5 )。
High-flow 100% normobaric hyperoxia prolongs penumbral survival in animals and appears to extend the time window for reperfusion therapy. 高血流量100%25正常气压高供氧延长了动物缺血半暗带的存活时间，且似乎可以延长再灌注治疗的时间窗。
